PLK4 is upregulated in prostate cancer and its inhibition reduces centrosome amplification and causes senescence - Singh - - The Prostate - Wiley Online Library
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PLK4 is upregulated in prostate cancer and its inhibition reduces centrosome amplification and causes senescence - Singh - - The Prostate - Wiley Online Library
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Retracted Article: MiR-148a agomir based targeting of c-Met and Her-3 is able to attenuate EGFR-T790M mutation driven gefitinib and erlotinib resistan ... - RSC Advances (RSC Publishing) DOI:10.1039/C8RA10224D
Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect
Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect
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PLK4 is upregulated in prostate cancer and its inhibition reduces centrosome amplification and causes senescence - Singh - - The Prostate - Wiley Online Library
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Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect
Cells | Free Full-Text | Elevated PDK1 Expression Drives PI3K/AKT/MTOR Signaling Promotes Radiation-Resistant and Dedifferentiated Phenotype of Hepatocellular Carcinoma | HTML
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Aβ-Induced Drp1 phosphorylation through Akt activation promotes excessive mitochondrial fission leading to neuronal apoptosis - ScienceDirect
PLK4 is upregulated in prostate cancer and its inhibition reduces centrosome amplification and causes senescence - Singh - - The Prostate - Wiley Online Library
PLK4 is upregulated in prostate cancer and its inhibition reduces centrosome amplification and causes senescence - Singh - - The Prostate - Wiley Online Library
Model D. Dosenfrosch - Publicaciones | Facebook
